Findings have important implications for the pathophysiology of key COVID symptoms and suggest new potential avenues for treatment
Not bad. I swear my cognitive functions have declined since contracting COVID, but it’s hard to prove and my GP blames it on age.
my GP blames it on age.
GPs are PFU.
In other words, the exact same secondary effect seen after the Flu pandemic 1918-20. People came down with encephalitis lethargica until the 1930s. Dopamine receptors were wiped out. The treatment was L-dopa, but not until 1961.
This sounds very interesting. Could you say a bit more/point to a good source to read more?
Long Covid is an umbrella disorder for lasting impacts of COVID, but the more debilitating outcomes that tend to get the attention in news articles are ME/CFS and autonomic disorders like POTS. Neither resemble encephalitis lethargica. New research into ME/CFS points to problems with neurons in the brain, but it’s not clear that symptoms are the result of irreversible damage. Patients sometimes go into remission and more commonly can regain some lost function with time and rest.
This has to be bittersweet for people with chronic fatigue syndrome or other post viral chronic conditions that are not long covid. On the one hand, it’s great that these conditions that have been just brushed aside for so long are finally getting some answers. On the other hand, limiting the research to just “long covid” continues to deny the fact that these symptoms have been known to be caused by a plethora of very common illnesses, and will likely limit care for that population.
As someone with ME, I’m mostly glad to see the research because we all know that they’re basically the same disease.
Great to hear about this. Does anyone know what medications boost the dopamine pathways since that was one of the venues mentioned in the article?
Levodopa, or L-dopa.
L-DOPA(also known as levo-dopa) i believe, but a doctor wouldnt prescribe you it though, used for early Parkinson’s symptoms.
Drugs used for Parkinson’s.
